Wang, Hu, Chen and colleagues published a Nature Communications paper demonstrating that mTORC1 signaling in group 2 innate lymphoid cells (ILC2s) couples neuro‑immune inputs to allergic lung inflammation. The team showed that mTORC1 activity modulates ILC2 effector function and the neuro‑immune axis that amplifies type 2 airway responses. ILC2s are innate immune cells that rapidly produce type 2 cytokines; the study clarifies a molecular node—mTORC1—that could be targeted to blunt neuro‑immune amplification in asthma and allergic disease.