Harvard-based cancer neuroscientist Humsa Venkatesh described how work on neural-tumor interactions has expanded beyond gliomas toward broader cancer types. Her earlier findings centered on electrochemical communication between neurons and tumor cells and identified a neuron-produced molecule implicated in glioma growth, which enabled testing of an inhibitor in a clinical trial. In follow-on research directions, Venkatesh emphasized a “reciprocal interaction” frame—how tumors remodel local nerves and how neural cues can promote progression. Coverage highlights how this concept may extend past the CNS, aligning with prior reports that peripheral nerves can innervate tumors such as prostate, gastrointestinal, and breast cancers. The updated focus also includes lung cancer, particularly in contexts where brain metastasis is common, underscoring efforts to map which cancers rely on neural signaling and how that biology could be therapeutically targeted.